By Privatdozent Dr. Faruk Hadžiselimović (auth.)
ISBN-10: 3642860206
ISBN-13: 9783642860201
ISBN-10: 3642860222
ISBN-13: 9783642860225
Ferment, an indication of development in any clinical box, has formerly been missing within the zone of cryptorchidism, the place the one task has been in bettering operative tools. Now, besides the fact that, profound alterations within the care of boys with cryptorchidism are being caused; rules are bobbing up from a clean examine comparative anatomy, and histological and experimental observa tions are being supplemented by means of medical exams made attainable by way of new hormonal brokers. The therapy of cryptorchidism starts with its reputation via the pediatri cian, who beforehand has proven little curiosity due to disappointing effects from chorionic gonadotropin management. As for the health practitioner, his bent towards recovery of standard anatomical relationships has stored his recognition desirous about the improvement of higher surgical technics to deliver the recalci trant testis into the scrotum. either experts have kept away from the primordial query of why the testis didn't descend correctly. If this have been identified they'd deal with the reason for cryptorchidism, and never be chuffed simply with attempting to right its consequence. As one reads this booklet, one sees that during such a lot sufferers cryptorchidism isn't as a result of a few anatomical constitution blocking off the best way or by means of a few deformity of the testis interfering with the delivery mechanism. fairly, deficiencies within the hormonal setting of the fetus retard the developmen tal sequences necessary to the traditional differentiation and descent of the testis. The scientific resolution is hence to supply the poor substances.
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Additional resources for Cryptorchidism: Management and Implications
Example text
The fibroblasts form concentric rings around the tubule. The interstitium contains mainly fetal Leydig cells, which are well developed and can be found in the interstitium until the 2nd year (Fig. 3 b; see also Chap. 3). 2 Fourth Year [1] The seminiferous tubule has an ultrastructural appearance quite different from that of a l-year-old. Gonocytes are no longer visible. In addition to the A-type spermatogonia, which can already be seen in the 1-year-old, B-type spermatogonia are encountered for the first time (Fig.
12). 021 spermatogonia/cm 3 testicular tissue in the cryptorchid testes, which is equivalent to a decrease in volume density of spermatogonia of approximately 60%. 005) (Table 1) [1 ]. 2 In Relation to Position The TFI (tubule fertility index) depends on the position at which the testis is retained. 005) [11]. 4%) [11]. However, on spermatogonia count per 50 tubule cross section there is a significant difference only between intra-abdominally and suprascrotally located testes (Table 2). It should be noted that all intra-abdominal testes do have germ cells withTable 2.
The partial isolated gonadotropin deficiency occurring in cryptorchid boys stops the transformation from A to B spermatogonia, as well as influencing to a certain extent multiplication of the germ cells. This is apparent if a congenitally cryptorchid gonad is compared to an iatrogenic cryptorchid gonad. Iatrogenic cryptorchidism resulting from hernia repair is one of the known complications [35]. The incidence given in the literature is less than 1 % (2/237 children [35]). In the last 10 years we had nine cases of iatrogenic cryptorchidism after hernia repair.
Cryptorchidism: Management and Implications by Privatdozent Dr. Faruk Hadžiselimović (auth.)
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