By Health Publica Icon Health Publications
ISBN-10: 0585482462
ISBN-13: 9780585482460
ISBN-10: 0597836302
ISBN-13: 9780597836305
It is a 3-in-1 reference ebook. It supplies a whole clinical dictionary protecting 1000s of phrases and expressions in terms of a number of sclerosis. It additionally offers large lists of bibliographic citations. ultimately, it presents details to clients on find out how to replace their wisdom utilizing a number of web assets. The e-book is designed for physicians, scientific scholars getting ready for Board examinations, clinical researchers, and sufferers who are looking to get to grips with study devoted to a number of sclerosis. in the event that your time is effective, this ebook is for you. First, you won't waste time looking out the web whereas lacking loads of proper info. moment, the ebook additionally saves you time indexing and defining entries. ultimately, you won't waste money and time printing hundreds and hundreds of web content.
Read or Download Multiple Sclerosis - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References PDF
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Additional resources for Multiple Sclerosis - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References
Sample text
Neurology; University of Rochester Orpa - Rc Box 270140 Rochester, NY 14627 Timing: Fiscal Year 2001; Project Start 01-JUN-2001; Project End 31-MAY-2005 Summary: Experimental autoimmune encephalomyelitis (EAE) is an autoimmune demyelinating disease of the central nervous system (CNS) induced by CD4+ T cells reactive with myelin antigens, including myelin basic protein (MBP). It is widely used as an animal model of multiple sclerosis (MS). There is growing evidence for a role of the Th1 polarizing monokine, IL-12, in the pathogenesis of EAE as well as MS.
Further, neonatal anti-TNF treatment completely prevents T1DM in NOD mice. In neonatal NOD mice, TNFalpha decreases the already low CD4+, CD25+ regulatory T cell population, while anti-TNF dramatically increases the number of these regulatory T cells; effects which could explain the unexpected results of these treatments. Several findings (23, 27) suggest that these effects may be due to the known ability of the TNFa p75 receptor (TNFR2) to mediate apoptosis in mature, activated, peripheral T cells (27).
Other groups have demonstrated the critical importance of newly rearranged CD4+ TCR alpha/beta+ regulatory T cells in preventing spontaneous EAE in TCR double transgenic mice that cannot produce regulatory T cells. It is thus hypothesized that TCR reactive T cells constitute the major portion of this important regulatory T cell population that thus far has not been characterized for antigen specificity. Two specific aims are proposed: Aim 1. Contribution of TCR-specific regulatory CD4+ T cells to EAE resistance.
Multiple Sclerosis - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References by Health Publica Icon Health Publications
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