By James Martin
ISBN-10: 0138511136
ISBN-13: 9780138511135
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29. Agarwal R. Blood pressure components and the risk for end-stage renal disease and death in chronic kidney disease. Clin J Am Soc Nephrol. 2009;4:830–7. 30. Champagne CM. Dietary interventions on blood pressure: the Dietary Approaches to Stop Hypertension (DASH) trials. Nutr Rev. 2006;64:S53–6. 31. Beyer KH. Chlorothiazide. How the thiazides evolved as antihypertensive therapy. Hypertension. 1993;22:388–91. 32. Report of the Joint National Committee on Detection, Evaluation, and Treatment of High Blood Pressure.
Stroke. Nov–Dec 1986;17(6):1126–8. 51. Zia E, Pessah-Rasmussen H, Khan FA, et al. Risk factors for primary intracerebral hemorrhage: a population-based nested case-control study. Cerebrovasc Dis. 2006;21(1–2):18–25. 52. Giroud M, Creisson E, Fayolle H, et al. Risk factors for primary cerebral hemorrhage: a population-based study—the Stroke Registry of Dijon. Neuroepidemiology. 1995;14(1):20–6. 53. Juvela S, Hillbom M, Palomaki H. Risk factors for spontaneous intracerebral hemorrhage. Stroke. Sep 1995;26(9):1558–64.
7) (refer Table 4). The risk of SAH increases sharply with the increase in systolic BP (78). This trend was also seen in a large cohort study among Asian populations for both men and women (Table 4) (62). Since most subarachnoid hemorrhages are caused by the rupture of a cerebral aneurysm, several studies have attempted to identify risk factors for the rupture/growth of an aneurysm. Family history, age and modifiable risk factors, including cigarette smoking, and hypertension have been thought to increase the risk of rupture (79–82).
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