By Stephen P. DiBartola DVM DACVIM
ISBN-10: 0721639496
ISBN-13: 9780721639499
The top reference for the analysis and administration of fluid, electrolyte, and acid-base imbalances in small animals, Fluid, Electrolyte, and Acid-Base problems in Small Animal perform, 4th Edition presents state of the art, evidence-based directions to augment your care of canine and cats. info is straightforward to discover and straightforward to exploit, with accomplished assurance together with fluid and electrolyte body structure and pathophysiology and their medical functions, in addition to the most recent advances in fluid remedy and a dialogue of a brand new classification of gear known as vaptans. Lead writer Stephen DiBartola is a widely known speaker and the "go-to" professional during this box, and his workforce of participants represents the main authoritative and revered clinicians and academicians in veterinary medication.
- Over 30 professional contributors characterize the "cream of the crop" in small animal medication, making sure that this variation presents the main authoritative and evidence-based guidelines.
- Scientific, evidence-based insights and advances combine uncomplicated physiological ideas into perform, masking sufferer review, differential prognosis, general and irregular medical gains and laboratory attempt effects, ways to remedy, technical facets of remedy, sufferer tracking, assessing hazard, and prediction of results for every affliction.
- Hundreds of tables, algorithms, and schematic drawings exhibit the easiest methods to analysis and therapy, highlighting crucial issues in an easy-access format.
- Drug and dosage recommendations are integrated with remedy ways within the Electrolyte Disorders section.
- Clear formulation within the Fluid Therapy section help you ensure the kingdom of dehydration, fluid selection, and management expense and quantity in either fit and diseased patients.
- Updated chapters hide the most recent advances in fluid treatment in sufferer management, supporting you realize and deal with a variety of very likely life-threatening metabolic disturbances.
- Expanded Disorders of Sodium and Water chapter contains details on a brand new classification of substances referred to as vaptans, vasopressin receptor antagonists that could quickly increase the facility to regulate sufferers with continual hyponatremia.
- Hundreds of latest references hide the main updated advances in fluid remedy, together with renal failure and surprise syndromes.
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Additional info for Fluid, Electrolyte, and Acid-Base Disorders in Small Animal Practice
Example text
In addition to its effects on insulin secretion, β-cell growth and proliferation, cAMP may also enhance differentiation. GLP-1 appears to increase the expression of pancreatic duodenal homeobox – 1 (PDX-1) (Perfetti et al. 2000), a transcription factor central to the differentiation of pancreatic endocrine, exocrine, and ductal cell populations from endoderm during fetal development (Zhou et al. 2002; Offield et al. 1996). PDX-1 is also necessary for maturation of β-cells and allows selective differentiation of pancreatic endocrine progenitors to form insulin-producing β-cells while suppressing formation of glucagon-producing α-cells (McKinnon and Docherty 2001; Ber et al.
Nat Methods 6(4):283–289. doi:10. 1313 Hnisz D, Abraham BJ, Lee TI, Lau A, Saint-Andre V, Sigova AA, Hoke HA, Young RA (2013) Super-enhancers in the control of cell identity and disease. Cell 155(4):934–947. 1016/j. 053 Hong SH, Ahmadian M, Yu RT, Atkins AR, Downes M, Evans RM (2014) Nuclear receptors and metabolism: from feast to famine. Diabetologia 57(5):860–867. 1007/s00125-0143209-9 Howald C, Tanzer A, Chrast J, Kokocinski F, Derrien T, Walters N, Gonzalez JM, Frankish A, Aken BL, Hourlier T, Vogel JH, White S, Searle S, Harrow J, Hubbard TJ, Guigo R, Reymond A (2012) Combining RT-PCR-seq and RNA-seq to catalog all genic elements encoded in the human genome.
PKA maintains temporal Role of the cAMP Pathway in Glucose and Lipid Metabolism 35 autoregulation of the cAMP signal while promoting lipolysis by allowing PDE3B fine-tuning of the cAMP signal to a concentration range that is ideal to sustain activity, whereas AKT blocks lipolysis by dissociating it from lipolytic signals at least in part by activating discrete pools of PDE3B. Significantly, PDE3B mutant mice show hepatic lipid accumulation and insulin resistance, suggesting that the increase in lipolysis is not accompanied by an increase in fatty acid oxidation and may reflect the redundancy between PDE family members or the diminished importance of PDE3B in the regulation of fatty acid oxidation in non-adipose tissues such as the liver.
Fluid, Electrolyte, and Acid-Base Disorders in Small Animal Practice by Stephen P. DiBartola DVM DACVIM
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